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p erbb3 tyr1289  (Cell Signaling Technology Inc)


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    Structured Review

    Cell Signaling Technology Inc p erbb3 tyr1289
    Reagents and tools table
    P Erbb3 Tyr1289, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 94/100, based on 126 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/p erbb3 tyr1289/product/Cell Signaling Technology Inc
    Average 94 stars, based on 126 article reviews
    p erbb3 tyr1289 - by Bioz Stars, 2026-02
    94/100 stars

    Images

    1) Product Images from "PlexinD1 is a driver and a therapeutic target in advanced prostate cancer"

    Article Title: PlexinD1 is a driver and a therapeutic target in advanced prostate cancer

    Journal: EMBO Molecular Medicine

    doi: 10.1038/s44321-024-00186-z

    Reagents and tools table
    Figure Legend Snippet: Reagents and tools table

    Techniques Used: Recombinant, Sequencing, Control, Transfection, Membrane, Reverse Transcription, Software, Microarray, Subcloning, shRNA, Cell Viability Assay, Viability Assay, Reporter Assay, In Situ, Enzyme-linked Immunosorbent Assay, Phospho-proteomics, Extraction, Bicinchoninic Acid Protein Assay, Magnetic Beads, Gel Extraction, Purification, Ligation, Mutagenesis, Chromatin Immunoprecipitation



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    Cell Signaling Technology Inc rabbit mab against p her3 y1289
    No significant changes of <t>p-HER3,</t> HER3, and p-FOXO3a were observed between DMSO- and lapatinib-treated tumors derived from SKBR3-pool2 or BT474-HR20 cells. The tumors derived from either SKBR3-pool2 ( A ) or BT474-HR20 ( B ) cells were formalin-fixed and paraffin-embedded (FFPE) and sectioned into five-micron-thick slides. The FFPE slides were analyzed with IHC staining assays for p-HER3 <t>(Y1289),</t> HER3, or p-FOXO3a (Ser253) following the procedures described in the materials and methods. Two individuals independently evaluated the IHC staining. The levels of p-HER3, HER3, and p-FOXO3a showed no apparent differences between control and lapatinib-treated groups
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    No significant changes of <t>p-HER3,</t> HER3, and p-FOXO3a were observed between DMSO- and lapatinib-treated tumors derived from SKBR3-pool2 or BT474-HR20 cells. The tumors derived from either SKBR3-pool2 ( A ) or BT474-HR20 ( B ) cells were formalin-fixed and paraffin-embedded (FFPE) and sectioned into five-micron-thick slides. The FFPE slides were analyzed with IHC staining assays for p-HER3 <t>(Y1289),</t> HER3, or p-FOXO3a (Ser253) following the procedures described in the materials and methods. Two individuals independently evaluated the IHC staining. The levels of p-HER3, HER3, and p-FOXO3a showed no apparent differences between control and lapatinib-treated groups
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    Image Search Results


    Reagents and tools table

    Journal: EMBO Molecular Medicine

    Article Title: PlexinD1 is a driver and a therapeutic target in advanced prostate cancer

    doi: 10.1038/s44321-024-00186-z

    Figure Lengend Snippet: Reagents and tools table

    Article Snippet: p-ErbB3 (Tyr1289) (WB: 1/1000; IHC: 1/1000) , Cell Signaling Technology , 2842.

    Techniques: Recombinant, Sequencing, Control, Transfection, Membrane, Reverse Transcription, Software, Microarray, Subcloning, shRNA, Cell Viability Assay, Viability Assay, Reporter Assay, In Situ, Enzyme-linked Immunosorbent Assay, Phospho-proteomics, Extraction, Bicinchoninic Acid Protein Assay, Magnetic Beads, Gel Extraction, Purification, Ligation, Mutagenesis, Chromatin Immunoprecipitation

    No significant changes of p-HER3, HER3, and p-FOXO3a were observed between DMSO- and lapatinib-treated tumors derived from SKBR3-pool2 or BT474-HR20 cells. The tumors derived from either SKBR3-pool2 ( A ) or BT474-HR20 ( B ) cells were formalin-fixed and paraffin-embedded (FFPE) and sectioned into five-micron-thick slides. The FFPE slides were analyzed with IHC staining assays for p-HER3 (Y1289), HER3, or p-FOXO3a (Ser253) following the procedures described in the materials and methods. Two individuals independently evaluated the IHC staining. The levels of p-HER3, HER3, and p-FOXO3a showed no apparent differences between control and lapatinib-treated groups

    Journal: Biological Procedures Online

    Article Title: Trastuzumab-resistant breast cancer cells-derived tumor xenograft models exhibit distinct sensitivity to lapatinib treatment in vivo

    doi: 10.1186/s12575-023-00212-3

    Figure Lengend Snippet: No significant changes of p-HER3, HER3, and p-FOXO3a were observed between DMSO- and lapatinib-treated tumors derived from SKBR3-pool2 or BT474-HR20 cells. The tumors derived from either SKBR3-pool2 ( A ) or BT474-HR20 ( B ) cells were formalin-fixed and paraffin-embedded (FFPE) and sectioned into five-micron-thick slides. The FFPE slides were analyzed with IHC staining assays for p-HER3 (Y1289), HER3, or p-FOXO3a (Ser253) following the procedures described in the materials and methods. Two individuals independently evaluated the IHC staining. The levels of p-HER3, HER3, and p-FOXO3a showed no apparent differences between control and lapatinib-treated groups

    Article Snippet: Primary antibodies used for immunohistochemistry (IHC) assays were the following: Rabbit mAb against p-Akt (Ser473) (Cat. #4060, 1:100 dilution), rabbit mAb against p-HER3 (Y1289) (Cat. #4791, 1:100 dilution), and rabbit mAb against HER3 (cat# 12708, 1: 400 dilution) from Cell Signaling Technology (Beverly, MA, USA); Rabbit polyclonal Ab against p-FOXO3a (Ser253) (Cat. #PA5-36816, 1:50 dilution) and mouse mAb against IRS1 (Cat. #MA5-36222, 1:50 dilution) from Thermo Fisher Scientific Inc. (Waltham, MA, USA).

    Techniques: Derivative Assay, Immunohistochemistry, Control